By Jeff Unger MD
This e-book deals basic care physicians evidence-based instructions for comparing and treating all sufferers with diabetes. It covers all facets of outpatient and hospital-based diabetes deal with all age teams, and comprises behavioral interventions for reinforcing sufferer adherence.Coverage contains fine-tuning glycemic administration with automated self-blood glucose tracking software program, comparing and treating sufferers with metabolic syndrome and polycystic ovary syndrome, dealing with psychological disease in sufferers with diabetes, and prevention of microvascular and macrovascular problems. The publication discusses more moderen insulin treatment protocols, insulin supply units similar to pumps, pens, and inhaled insulin, and novel pharmacotherapies together with incretin mimetics and DPP-IV inhibitors.
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Wu A, Tseng C-C, Van Den Berg D, Yu MC: Tea intake, COMT genotype and breast cancer in Asian-American women. Cancer Res 2003;63:7526–7529. Dr. A. El-Sohemy Department of Nutritional Sciences, Faculty of Medicine University of Toronto, 150 College St. Toronto, Ont. M5S 3E2 (Canada) Tel. ca El-Sohemy 30 Tai ES, Gillies PJ (eds): Nutrigenomics – Opportunities in Asia. Forum Nutr. Basel, Karger, 2007, vol 60, pp 31–41 Epigenomics and Nutrition Lynne Cobiac Preventative Health National Research Flagship, CSIRO, Adelaide, Australia Abstract Epigenomics or epigenetics refers to the modification of DNA that can influence the phenotype through changing gene expression without altering the nucleotide sequence of the DNA.
Dr. John A. Milner Nutritional Science Research Group, Division of Cancer Prevention National Cancer Institute, 6130 Executive Boulevard, Suite 3164 Rockville, MD 20892 (USA) Tel. gov Milner 24 Tai ES, Gillies PJ (eds): Nutrigenomics – Opportunities in Asia. Forum Nutr. , Canada Abstract Nutrients interact with the human genome to modulate molecular pathways that may become disrupted, resulting in an increased risk of developing various chronic diseases. Genetic polymorphisms affect the metabolism of dietary factors, which in turn affects the expression of genes involved in a number of important metabolic processes.
Do we need to revisit some of the nutrient reference values where in some instances it appears that there are much higher levels of for example folate needed to maintain genome stability ? Nutrients may have a protective effect on one pathway and a nonprotective effect on another and these complex interplays need to be understood. Furthermore, nutrients or bioactives may be protective at one level and nonprotective at a higher level and this effect may depend on the genetic background of the individual.