By Roy Porter
"Chock-full of magnificent proof and interesting illustrations."—BooklistAn eminently readable, unique romp during the heritage of our useless and valiant efforts to heal ourselves. Mankind's conflict to stick alive and fit for so long as attainable is our oldest, such a lot common fight. together with his attribute wit and tremendously knowledgeable historic scope, Roy Porter examines the battle fought among disorder and medical professionals at the battleground of the flesh from precedent days to the current. He explores the various creative ways that we have now tried to beat affliction in the course of the a long time: the altering position of medical professionals, from historic healers, apothecaries, and blood-letters to today's pros; the array of substances, from Ayurvedic treatments to the release of Viagra; the advances in surgical procedure, from amputations played via barbers with out anesthetic to today's subtle transplants; and the transformation of hospitals from Christian locations of convalescence to fashionable scientific powerhouses. Cleverly illustrated with old line drawings, the continual diseases of humanity supply vibrant anecdotes for Porter's enlightening tale of medicine's efforts to be successful over an impressive and ever-changing adversary.
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Extra resources for Blood and Guts: A Short History of Medicine
In patients with less than 1% activity, frequent hemarthroses and severe bleeding episodes develop. The diagnosis is suggested by an elevated PTT, normal PT, and normal bleeding time. Factor activity assays confirm the diagnosis. a. Treatment of hemophilia A is based on the degree of bleeding and the severity of the disease. 1. Minor bleeding often is controlled locally without factor replacement therapy. ) stimulates the release of vWF, which complexes with factor VIII, thereby sheltering it from the circulation.
B. Diagnosis. Although the cause of metabolic alkalosis is usually apparent in the surgical patient, measurement of urinary chloride concentration may be useful for differentiating these disorders. Urine Cl – concentration of less than 15 mmol/L suggests inadequate fluid resuscitation, ongoing GI loss from emesis or nasogastric suctioning, diuretic administration, or posthypercapnia as the cause of the metabolic alkalosis. Urine Cl – concentration greater than 20 mmol/L suggests mineralocorticoid excess, alkali loading, concurrent diuretic administration, or the presence of severe hypokalemia.
Acquired a. Lupuslike inhibitors are IgG, IgA, or IgM immunoglobulins that are targeted against antigens composed in part of platelet and endothelial cell phospholipids. , chlorpromazine, procainamide, or hydralazine). Approximately half of patients with lupuslike inhibitors have no identifiable predisposing conditions and are said to have antiphospholipid antibody syndrome. The diagnosis is suggested by a prolonged PTT and can be confirmed using specific tests, such as Russell's viper venom clotting time (for lupus anticoagulant) and anticardiolipin antibody immunoassay.