Download BarCharts QuickStudy Lymphatic System by Inc. BarCharts PDF

By Inc. BarCharts

Complete, categorized illustrations of the components of the lymphatic approach in 9 various perspectives and sections.

From pre-teen to pre-med, this chart is loaded with superbly illustrated diagrams in actual fact and concisely categorized for simple identity.

Illustrations via award-winning clinical illustrator Vincent Perez.

Chart contains targeted diagrams of:
· head & neck
· arm axilla & thorax
· nodes & vessels
· thoracic duct
· deep belly & inguinal nodes
· huge gut
· belly & pancreas
· center & lungs

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1995) Obesity in heterozygous carriers of the gene for the Bardet–Biedl syndrome. American Journal of Medical Genetics, 55, 12–15. 16. , Durand, E. et al. (2000) Melanocortin-4 receptor mutations are a frequent and heterogeneous cause of morbid obesity. Journal of Clinical Investigation, 106, 253–62. 17. , Rankinen, T. et al. (2002) Melanocortin 4 receptor sequence variations are seldom a cause of human obesity: the Swedish Obese Subjects, the HERITAGE Family Study, and a Memphis cohort. Journal of Clinical Endocrinology and Metabolism, 87, 4442–6.

Thus, the many benefits of performing genetic studies in mice may be reaped whilst maintaining relevance to human populations. These advantages include stable environmental conditions, pure breeding strains, the ability to perform sibling and back-cross mating, rapid breeding (generation) times and the ability to obtain accurate phenotypical data. The use of this method has identified QTLs having effects on intermediary phenotypes such as thermogenesis, serum leptin levels and insulin resistance, gene-environment interactions such as susceptibility to diet-induced obesity and with time of obesity onset and gender-specific susceptibility to obesity.

The principal advantage of this methodology is that interactions between loci each having only a small phenotypic effect may be investigated particularly where the sum of more than one such effect is large. The principle of attempting to define QTLs as a tool for gene mapping relies on the fact that rodents may be selectively inbred to produce strains that differ widely in body weight and fat deposition under the influence of polygenic loci. Breeds with opposite poles of a phenotypic extreme may then be cross bred to produce F1 progeny whose phenotype will depend on the exact genotype inherited from each parental strain [34].

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